Receptor tyrosine kinase-dependent PI3K activation is an escape mechanism to vertical suppression of the EGFR/RAS/MAPK pathway in KRAS-mutated human colorectal cancer cell lines https://t.co/6KWlWg40W2 … #hvhebron #onco #ScientiaVH @BioMedCentral https://t
A work by my collegue Pietro Paolo Vitiello about role of PI3K activation in acquired resistance to vertical suppression of EGFR/MAPK pathway in KRAS mutated CRC https://t.co/w8CJ0GjF6V
Very proud to see finally published experimental results of my dear colleague and friend Pietropaolo Vitiello about KRAS mutated colorectal cancers!!! https://t.co/V6o2leSd5C #
RT @BibliotecaHUVH: Receptor tyrosine kinase-dependent PI3K activation is an escape mechanism to vertical suppression of the EGFR/RAS/MAPK…
Receptor tyrosine kinase-dependent PI3K activation is an escape mechanism to vertical suppression of the EGFR/RAS/MAPK pathway in KRAS-mutated huma... https://t.co/4itTL36ezl #hvhebron #onco @OncoBioMed [Text complet]