Methylglyoxal-derived advanced glycation end products contribute to negative cardiac remodeling and dysfunction post-myocardial infarction

Overview of attention for article published in Basic Research in Cardiology, September 2017

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https://t.co/tUIQqd6bIM ⚠️ Therefore, inhibiting > MAPK, NLRP3 Inflammasome, AGEs and mTOR > decreases ACS complications 💊MK > inhibits CysLT1R > inhibits mTOR and AGEs p38MAPK > ⬇️ microglial activity NF-ΚB > ⬇️ neuropathic pain NL

21 Aug 2023

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Methylglyoxal-derived advanced glycation end products contribute to negative cardiac remodeling and dysfunction post-myocardial infarction. https://t.co/rsVTh1XtWV

22 May 2018

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メチルグリオキサール由来のＡＧＥは心筋梗塞後心臓のリモデリングに対してネガティブな影響を与える。ラットのモデル。#AGE https://t.co/AwSpbJSnjn

23 Sep 2017

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Newest publication from the UOttawa Dept of #Surgery and @HeartInstitute Congratulations Dr. Suuronen. https://t.co/ryrDB7eSFL https://t.co/3eItdqUgJo

18 Sep 2017

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